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complex diseases. New wet-lab or in-silico methods
to accurately reconstruct very long haplotypes instead
of using the expensivenuclear family data sets haveto
be defined. Moreover, the method needs to be tested
in other polygenic diseases.
ACKNOWLEDGEMENTS
The authors were supported by the Spanish Research
Program under project TIN2010-20900-C04, the An-
dalusian Research Program under project P08-TIC-
03717 and the European Regional Development Fund
(ERDF). The authors thank Paola Sebastiani for her
help in the work undertaken.
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