Research of Inducing Apoptosis and Ferroptosis in Ovarian Cancer and Its Synergistic Anticancer Significance
Qian Yang
2022
Abstract
Ovarian cancer (OC), as a highly malignant tumor of female reproductive system, has the highest mortality. At present, in the clinical treatment of OC, although the action mechanism of some chemical drugs is clear and the curative effect is remarkable, they are easy to result in drug resistance, which seriously hinders the anticancer effect of chemotherapeutic drugs. In this brief article, we summarized the two cell death modes of apoptosis and ferroptosis, discussed the correlation between apoptosis, ferroptosis and OC, and emphasized the synergistic effect of combined application of drugs that can induce cell apoptosis and ferroptosis to deal with the drug resistance of OC. The latest progress of this subject will help to deal with the drug resistance of OC and expand more ideas for clinical treatment.
DownloadPaper Citation
in Harvard Style
Yang Q. (2022). Research of Inducing Apoptosis and Ferroptosis in Ovarian Cancer and Its Synergistic Anticancer Significance. In Proceedings of the 4th International Conference on Biotechnology and Biomedicine - Volume 1: ICBB; ISBN 978-989-758-637-8, SciTePress, pages 223-227. DOI: 10.5220/0012018700003633
in Bibtex Style
@conference{icbb22,
author={Qian Yang},
title={Research of Inducing Apoptosis and Ferroptosis in Ovarian Cancer and Its Synergistic Anticancer Significance},
booktitle={Proceedings of the 4th International Conference on Biotechnology and Biomedicine - Volume 1: ICBB},
year={2022},
pages={223-227},
publisher={SciTePress},
organization={INSTICC},
doi={10.5220/0012018700003633},
isbn={978-989-758-637-8},
}
in EndNote Style
TY - CONF
JO - Proceedings of the 4th International Conference on Biotechnology and Biomedicine - Volume 1: ICBB
TI - Research of Inducing Apoptosis and Ferroptosis in Ovarian Cancer and Its Synergistic Anticancer Significance
SN - 978-989-758-637-8
AU - Yang Q.
PY - 2022
SP - 223
EP - 227
DO - 10.5220/0012018700003633
PB - SciTePress